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      Environmental Inequality in Exposures to Airborne Particulate Matter Components in the United States

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          Abstract

          Background: Growing evidence indicates that toxicity of fine particulate matter ≤ 2.5 μm in diameter (PM 2.5) differs by chemical component. Exposure to components may differ by population.

          Objectives: We investigated whether exposures to PM 2.5 components differ by race/ethnicity, age, and socioeconomic status (SES).

          Methods: Long-term exposures (2000 through 2006) were estimated for 215 U.S. census tracts for PM 2.5 and for 14 PM 2.5 components. Population-weighted exposures were combined to generate overall estimated exposures by race/ethnicity, education, poverty status, employment, age, and earnings. We compared population characteristics for tracts with and without PM 2.5 component monitors.

          Results: Larger disparities in estimated exposures were observed for components than for PM 2.5 total mass. For race/ethnicity, whites generally had the lowest exposures. Non-Hispanic blacks had higher exposures than did whites for 13 of the 14 components. Hispanics generally had the highest exposures (e.g., 152% higher than whites for chlorine, 94% higher for aluminum). Young persons (0–19 years of age) had levels as high as or higher than other ages for all exposures except sulfate. Persons with lower SES had higher estimated exposures, with some exceptions. For example, a 10% increase in the proportion unemployed was associated with a 20.0% increase in vanadium and an 18.3% increase in elemental carbon. Census tracts with monitors had more non-Hispanic blacks, lower education and earnings, and higher unemployment and poverty than did tracts without monitors.

          Conclusions: Exposures to PM 2.5 components differed by race/ethnicity, age, and SES. If some components are more toxic than others, certain populations are likely to suffer higher health burdens. Demographics differed between populations covered and not covered by monitors.

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          Most cited references90

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          Environmental Justice

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            Health effects of fine particulate air pollution: lines that connect.

            Efforts to understand and mitigate thehealth effects of particulate matter (PM) air pollutionhave a rich and interesting history. This review focuseson six substantial lines of research that have been pursued since 1997 that have helped elucidate our understanding about the effects of PM on human health. There hasbeen substantial progress in the evaluation of PM health effects at different time-scales of exposure and in the exploration of the shape of the concentration-response function. There has also been emerging evidence of PM-related cardiovascular health effects and growing knowledge regarding interconnected general pathophysiological pathways that link PM exposure with cardiopulmonary morbidiity and mortality. Despite important gaps in scientific knowledge and continued reasons for some skepticism, a comprehensive evaluation of the research findings provides persuasive evidence that exposure to fine particulate air pollution has adverse effects on cardiopulmonaryhealth. Although much of this research has been motivated by environmental public health policy, these results have important scientific, medical, and public health implications that are broader than debates over legally mandated air quality standards.
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              Environmental Health Disparities: A Framework Integrating Psychosocial and Environmental Concepts

              Although it is often acknowledged that social and environmental factors interact to produce racial and ethnic environmental health disparities, it is still unclear how this occurs. Despite continued controversy, the environmental justice movement has provided some insight by suggesting that disadvantaged communities face greater likelihood of exposure to ambient hazards. The exposure–disease paradigm has long suggested that differential “vulnerability” may modify the effects of toxicants on biological systems. However, relatively little work has been done to specify whether racial and ethnic minorities may have greater vulnerability than do majority populations and, further, what these vulnerabilities may be. We suggest that psychosocial stress may be the vulnerability factor that links social conditions with environmental hazards. Psychosocial stress can lead to acute and chronic changes in the functioning of body systems (e.g., immune) and also lead directly to illness. In this article we present a multidisciplinary framework integrating these ideas. We also argue that residential segregation leads to differential experiences of community stress, exposure to pollutants, and access to community resources. When not counterbalanced by resources, stressors may lead to heightened vulnerability to environmental hazards.
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                Author and article information

                Journal
                Environ Health Perspect
                EHP
                Environmental Health Perspectives
                National Institute of Environmental Health Sciences
                0091-6765
                1552-9924
                10 August 2012
                December 2012
                : 120
                : 12
                : 1699-1704
                Affiliations
                [1]School of Forestry and Environmental Studies, Yale University, New Haven, Connecticut, USA
                Author notes
                Address correspondence to M.L. Bell, School of Forestry and Environmental Studies, Yale University, 195 Prospect St., New Haven, CT 06511 USA. Telephone: (203) 432-9869. Fax: (203) 436-9135. E-mail: michelle.bell@ 123456yale.edu
                Article
                ehp.1205201
                10.1289/ehp.1205201
                3546368
                22889745
                3394f3bb-6cc9-41bd-9d5c-26caf296c2c3
                Copyright @ 2012

                This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, properly cited.

                History
                : 12 March 2012
                : 10 August 2012
                Categories
                Research

                Public health
                air pollution,chemical components,environmental justice,particulate matter,pm2.5,race,socioeconomic status

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